At present, any “connection” between these two events is “Rank Speculation”. There is no “there there” in terms of evidence. This is just at the start of “connect the dots” where you have two dots and no idea if there are any more, or if these two even connect.
The “effect” in question would be a slower / delayed onset of autoimmune disease, especially in older folks or those with some preexisting predisposition.
What is a known fact is that for some folks the Chinese Wuhan Covid disease and the Genetic Frankenshot injection can cause various immunity mediated problems including things like thrombocytopenia and neuropathy.
It is also known that the injections cause your cells to produce spike proteins and release them into the blood AND that the spike protein itself is toxic to cells, causes damage, and instigates the intense immune response. The worst damage from Covid-19 comes when the virus population is dropping, but the virus fragments are floating around in the blood causing cytokine storms, doing damage, and causing in inflamation spike.
This means that the injection itself ought to cause many of the same symptoms and effects as a Chinese Wuhan Covid infection; just hopefully for most people at a lower level of intensity.
But will it be lower for everyone?
If you suffer from neuropathy caused by an autoimmune disease and need regular blood infusions, be aware that most blood donors have not been tested for COVID-19 antibodies. If you’re aware of the risks related to your neuropathy, you can adjust and protect yourself.
If you do become infected with the virus, you’re not likely to experience any new damage to your cells, but you may have flare-ups of your neuropathic symptoms.
The flu-like effects of COVID-19 may exacerbate the tingling and numbness you normally feel. While this may be uncomfortable, it’s no need to panic. Follow your doctor’s care orders closely until the infection runs its course.
So why are we NOT screening blood supply for Covid-19 antibodies? Seems like it would give a good insight into the status of “Herd Immunity” and / or virus prevalence.
But notice that it is known that Chinese Wuhan Covid will make any peripheral neuropathy worse.
Neurologic Symptoms in COVID-19 patients
The most common neurologic symptom reported is loss of smell, which may occur early in the disease process. Loss of smell, which may also impact the sense of taste, is thought to be temporary and it is felt that most people recover their sense of smell over time. Some of the latest research would indicate that this is because of inflammatory changes affecting not the olfactory neurons (which receive and transmit odors from the nose to the brain), but rather the support cells that lead to a healthy nasal mucosa and healthy olfactory neurons. Aside from waiting, there is no definite treatment to help recover the sense of smell, but some have suggested that taking oral omega-3 supplements or using intranasal vitamin A may help speed recovery.
Medicine by “how I feel about the disease”? Hoping the sense of smell will recover just fine? OK… But clearly this virus (and the spike proteins) attack nerves in the nose.
Guillain-Barré syndrome is an inflammatory disorder affecting the peripheral nerves and the nerve roots that can lead to generalized muscle weakness, numbness, walking problems, and in severe cases affect breathing, blood pressure, and the gastrointestinal system. Prior to COVID-19, Guillain-Barré syndrome had been linked to an inflammatory response as a result of several infections, including Campylobacter jejuni, Lyme disease, EBV, HIV, and Zika virus, to name a few. Almost expectedly, there are now many reports of Guillain-Barré syndrome occurring during the acute illness from SARS-CoV-2 infection and within the weeks following recovery. Fortunately, should this happen it seems that the standard treatments for Guillain-Barré syndrome are seen to be beneficial, and most patients recover very well over time and with physical therapy.
Again an attack on nerve structures. Note particularly that it can be weeks following recovery, not just “in the moment” of the disease. This also implies it can be in the “weeks following” the Frankenshot too. Expecting things to happen “in the now” or attempting to use “it happened weeks after” to say it was not caused by the virus or the shot is a mistake.
Much more commonly, however, we are seeing many patients in our practices complain of mild but still disabling muscle weakness, fatigue, and numbness or tingling following COVID-19 disease. When we see these patients, we often recommend a series of standard tests looking for neuropathy or myopathy. Checking certain blood tests is important to identify other causes of neuropathy that may be contributing to these symptoms, such as vitamin deficiencies or metabolic derangements such as diabetes. Nerve conduction studies and electromyography (EMG) testing may be performed to characterize the presence of a neuropathy. Making a formal diagnosis is challenging in many of these patients, however, because the nerve conduction studies and EMG are often normal relative to the standard reference values in our experience. In addition, skin biopsies which look for evidence of damage to or loss of the small nerve fiber endings in the skin are often normal as well, though there have been no formal rigorous studies yet to confirm this.
While we are also anecdotally seeing a major uptick in people reporting new or worsening of their headaches or migraines following COVID-19 disease, these symptoms seem to improve with time and with traditional recommendations for headache. Probably, the most debilitating symptoms reported by patients is the overall feeling of fatigue, mental fog, and quantifiable weight loss in the weeks and months following the infection. Especially among people who live in New York City where we practice, the daily stresses of life can be overwhelming on a good day. Many people who had COVID-19 are now reeling from this major drain on both their physical and mental energy. This is compounded by the overall level of physical deconditioning that many New Yorkers are facing by quarantining indoors the past six months, from the closure of most gyms, and the inability to engage in many other physical activities. Unfortunately, there has yet to be a label applied to this condition, no clear diagnostic tests, and no definitive treatments identified.
Peripheral Neuropathy and COVID-19
Neuropathic pain is not an uncommon post-Covid symptom. However, we don’t have definite evidence to prove that the worsening of the neuropathy symptoms is really from peripheral neuropathy – or neuropathy like symptoms coming from problems with the brain or spinal cord. Anecdotally some patients with these symptoms show no large or small fiber neuropathy on EMG and skin biopsies. The symptom management however is similar, and we again expect to learn more with time.
(You can read an article written by FPN’s Board Member that appeared in Brain and Life magazine tells his journey with COVID-19 and peripheral neuropathy.)
Now I would expect that if someone who has no prior problem can get peripheral neuropathy problems, then someone with it already can get worse. I’d further expect that since the spike protein itself is mediating for the problem, a shot that causes production and blood circulation of the spike protein could also cause the problem. Perhaps at a lower level, but enough to cause a person who is already dealing with peripheral neuropathy or autoimmune disorders to have them worsen. Yes, speculative, but informed speculation and something that ought to be investigated, not swept under the rug with “just a side effect”… that can show up weeks later and take months to resolve.
Ambient respiratory viral infections have been associated with an increased number of cases of rheumatoid arthritis (especially in women and older patients), suggesting that respiratory viral infections might be an environmental risk factor for the development of rheumatoid arthritis.
SARS-CoV-2 enters the cell via the angiotensin-converting enzyme-2 (ACE-2) and it is sensed by Toll-like receptor-7 (TLR7); bioinformatic analyses have shown that the SARS-CoV-2 genome contains a large number of fragments that are recognised by TLR7. In addition to its expression in immunological cells, TLR7 is expressed predominantly in the lung and bronchus, thus allowing SARS-CoV-2 to be highly recognised in the regions of its tropism. TLR7 activation leads to activation of c-Jun N-terminal kinase and nuclear factor-κB signaling, thus leading to the production of IL-6 and IL-12p40. 5 Therefore, it is conceivable that patients with COVID-19 might display symptoms and signs of inflammation, such as a viral arthritis.6 Thus, from the point of view of a rheumatologist, evaluating the role of SARS-CoV-2 in inflammatory arthritis is essential for diagnosis.
Serological tests could be useful to establish a diagnosis, but the possibility that low-titre positivity for autoantibodies (such as rheumatoid factor [RF] or antinuclear antibody [ANA]) could be detected in viral arthritis must also be taken into account.
A detailed analysis of epidemiological, clinical, and serological characteristics is required to help physicians diagnose viral arthritis; oligoarticular or polyarticular involvement (either symmetric or asymmetric), good response to NSAIDs, a clinical manifestation characterised by an early onset (within the first weeks of symptomatic infection) and a self-limiting presence are the elements that orientate toward a viral arthritis (appendix p 1).
Note that simply binding to the receptor is enough to trigger the “sensing” and cascade of effects. The spike protein binds to the receptor.
OK, that’s the disease. We’re talking about the so called “vaccination” shot (that is really a “genetic device” not a classical vaccine). Any evidence that it’s doing the same thing? Other than rank speculation that “vaccine” produced spike protein will act the same way as virus produced spike protein?
Here, we present a case of a White male, aged 55 years, with non-erosive, seropositive rheumatoid arthritis (positive for rheumatoid factor, anticyclic citrullinated peptide antibodies, antinuclear antibodies, and anti-Ro antibodies) who had been in sustained clinical remission for more than 2 years and developed an acute flare of his rheumatoid arthritis 12 h after the second BNT162b2 vaccination.
The patient had been in clinical remission on upadacitinib monotherapy since July, 2018. At his last clinic visit in September, 2020, his physical exam showed no active synovitis or joint effusions, and his disease activity scores were consistent with remission (clinical disease activity index=0; disease activity score of 28 joints with C-reactive protein=1·21). The patient had no known exposures to SARS-CoV-2 and had tested negative for SARS-CoV-2 by PCR in April, 2020, when screened for work. He received the first BNT162b2 vaccine on Dec 23, 2020, after which he developed minor arthralgias that resolved within 1 day. He received the second vaccine dose on Jan 13, 2021, and within 12 h developed clinically significant pain and swelling in the right knee. He had no other joint pain, swelling, or stiffness. He took ibuprofen and prednisone 5 mg soon after the pain and swelling began, but his symptoms persisted, so he contacted our office the next day and we advised him to increase his prednisone to 10 mg daily. Despite the increased medication, he continued to have clinically significant symptoms, so we asked him to come to our clinic for an ultrasound evaluation. He had continued on his usual rheumatoid arthritis treatment between the two vaccinations.
The patient came to our clinic on Jan 22, 2021—30 days after receiving the first BNT162b2 vaccination, and 9 days after the second vaccination—he had clinically significant swelling and warmth over the right knee with pain on flexion and extension of the knee. There was no tenderness, swelling, or erythema of any other joints. Ultrasound evaluation of the right knee showed a moderate to large compressible hypoechoic effusion in the suprapatellar recess that extended from the suprapatellar bursa proximally 5·2 cm deep to the quadriceps tendon and involved the medial and lateral gutter (appendix). There was increased power Doppler signal in the effusion in the lateral gutter. Additionally, there was a large effusion in the posterior knee deep to the semimembranosus tendon, consistent with a popliteal cyst.
His rheumatoid arthritis was well controlled before the vaccination, and there were no other inciting events, so we believe that this flare might have been triggered by his immune response to a component of the BNT162b2 vaccine. BNT162b2 contains mRNA encoding for the SARS-CoV-2 spike protein encapsulated in lipid nanoparticles, in addition to other components that stabilise the vaccine in the circulation and promote its uptake into cells by endocytosis. Although the mechanism of flare is not known, one could speculate that one of these components might have had non-specific adjuvant effect, or there could have been molecular mimicry between the viral spike peptides and the patient’s self-peptides, activating a flare. However, we cannot exclude the possibility that the timing of the flare with regard to vaccination was coincidental. The patient was treated with intra-articular steroids with rapid improvement, and he is once again in clinical remission.
So I’d count that as a “yes”. Poke the bear, it wakes up sometimes. As people are highly variable, I’d expect many to have zero problems, some to have modest problems, and some to be debilitated.
Which brings us to the 2 data points.
(h/t to P.G. Sharrow for this one, here:)
Eric Clapton discusses his experience with taking the Covid Jab;
Be sure to watch the video link half way down in the article.
In the article we have Clapton saying:
Repeating his previously-stated scepticism about the coronavirus vaccine, Clapton said he received his for the sake of his children, but that side effects then left him “out for the count for about a week,” and severely worsened his existing peripheral neuropathy, leading to “agony” and “chronic pain.”
He added that the second dose left him unable to use his hands for three weeks, and that he still “can’t touch anything cold or hot” without the use of gloves.
When your entire career and skill are in your hands, that’s pretty devastating. Not to mention the persistent pain he is dealing with.
Here’s the video:
Which brings me to the second data point.
Over the years I’ve frequently pointed at iceagenow.info as a site that does a great job with reporting the abnormal cold and snow weather events. Seems to me (though not to him) that the blog operator, Robert, has had a similar brush with the Frankenshot:
Published June 8, so about a week ago. There have been no knew articles since, and no update of comments since. I have at least 2 that have not yet made it through “moderation”. My belief is that (quite properly) Robert is dealing with his health issues and letting the blog sit for a while.
But what are they? As he puts it, he doesn’t know, nor do we. He asserts there is no connection to his vaccination as there was a time gap between them, but we know that a delayed response is common. So maybe yes, maybe no.
You’ll want to read this if you have rheumatoid arthritis (R.A.). You also may want to share this with any of your friends or relatives who have R.A.
For starters, I simply do not know how I’m doing!
Fast forward to 2018, when I needed to undergo a quite invasive surgery. The surgeon asked me to go off all meds, including the methotrexate, for 6 weeks prior to the surgery, and six weeks after.
Lo-and-behold, the R.A. had vanished! Gone! I thought. My rheumatologist and I decided to discontinue the methotrexate, and after awhile he dismissed me. Told me to call him in the event the R.A if I have a problem.
Bad, bad, bad mistake.
So now we get to today.
Two Thursdays ago I walked into my first meeting with a rheumatologist because of a long list of symptoms that specialists couldn’t fathom. None of the specialists had ever mentioned R.A. Not once. Finally, after months, my wife and I self-diagnosed the culprit as R.A. The rheumatologist agreed. Hugely progressive R.A. By Friday night I was walking with a cane. By Sunday night I needed a walker. Now I’m dependent on a wheelchair. All in less than three weeks.
Went to the JPS Hospital emergency room. The hospital infused me with a high dose of steroids (1000 mg of prednisone) three days in a row which was supposed to stop the progression in its tracks. It did nothing.
They couldn’t believe R.A. could work that quickly, so they conducted a full-body MRI, a cat-scan of my head, a Lumbar puncture (spinal tap) and so many blood tests that I couldn’t possibly keep count. Nothing. They’re completely baffled.
In the meantime I’m sticking with my belief that it’s R.A. because my rheumatoid factor is 50. (About 4 times what it should be.) Instead of attacking my joints, it is attacking my organs – my skin (an organ), heavy neuropathy, numbness in feet and lower legs (my legs have turned to jello and I have no balance), and the numbness has begun moving into my hands. Highly elevated blood pressure, highly elevated liver enzyme levels, the list goes on and on.
And oh, did I mention the pain?
Now we’re waiting for the insurance company to hopefully approve treatment with a drug called Rituxan. (Chemotherapy).
In the meantime I’m home from the hospital but getting progressively worse. I’m taking as many pain meds as they’ll prescribe.
All in less than three weeks!
I’ll keep you updated, and please wish me good luck.
I’ve bolded the point about 3 weeks of rapid accelleration.
Further down, in comments:
June 8, 2021 at 3:11 pm
Were you Covid-vaxxed?
June 8, 2021 at 3:24 pm
Yes. J&J, several weeks ago. But please understand, this has been going on since at least last July.
Yes, ambiguous. Is it just a natural acceleration of a return of RA? Or did the vaccination stomp on the “Go Pedal” for the immune response?
That he had a variety of unusual non-RA symptoms prior to getting the RA panel of tests, and then the rapid, I’d even say dramatic, acceleration of issues, to me, speaks to something outside of the usual RA patterns.
But like I said, just 2 data points and just rank speculation.
I hope things go well for Robert. I’ve valued his contributions over the years on the Global Warming fraud. I would hate to see his voice stilled by the Chinese Wuhan Covid plandemic and / or the Frankenshot.
Yes, I’m a bit peeved about all this.
FWIW, my suggestion is that folks with existing autoimmunity issues consider using some kind of prophylactic instead of an immune poking spike protein surge. Frankly, my own tendency to “high immune response” is why, about a year ago, I decided I wanted immune soothing prophylactics rather than immune poking mRNA Spike Protein production inside my body. But that is just my personal choice.
Should I ever get the “vaccination” shot, I will be dosed with Ivermectin prior to it. Ivermectin has been shown to bind to the end of the spike protein and help block binding to the ACE-2 receptor. It is my speculation that this would help prevent a hyper-immunity cascade from the shot as that whole metabolic cascade with TLR7 would be muted or prevented. Yet the proteins would still be floating around where the immune system could sensitize to them.
In short, I think it has a good chance of preventing vaccination side effects similar to how it prevents disease symptoms.
For now, we’ve got 2 data points, some medical article “one off” cases, and some rank speculation. But I think it is enough to say “Watch Here!” for now and eventually “Dig Here!” when more cases show up.